By Author Unknown
The sphere of mitochondrial illnesses is presently one of many swiftly transforming into fields of analysis in cellphone and molecular biology. This quantity encompasses the most recent improvement during this box of analysis. The chapters disguise subject matters in a variety of disciplines together with biophysics, biochemistry, mobilephone and molecular biology, molecular genetics, and scientific drugs.
Summarizes starting to be proof of the function of mitochondria in a lot of pathological conditions
Brings jointly diversified techniques towards figuring out mitochondria diseases
Molecular and mobile biology
Clinical body structure and medicine
Details the an important function this organelle performs in genetic law of varied organic services.
Read or Download Current Topics in Bioenergetics, Volume 17: Molecular Basis of Mitochondrial Pathology PDF
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Additional resources for Current Topics in Bioenergetics, Volume 17: Molecular Basis of Mitochondrial Pathology
And Hegner, D. (1978). Eur. J. Biochem. 82, 563-567. , and Hegner, D. (1978). Eur. J. Biochem. 90, 385-390. , and Summer, Κ. H. (1989). Free Radical Biol. Med. 6, 369-374. Osiewacz, H. D. (1990). Mutat. Res. 237, 1-8. , and Horai, S. (1988). Biochem. Biophys. Res. Commun. 154, 1240-1247. , and Sahashi, K. (1990a). In "Bioenergetics" (C. H. Kim and T. ), pp. 413-427. Plenum, New York. , and Mizuno, Y. (1990b). Biochem. Biophys. Res. Commun. 172, 483-489. , and Yoneda, M. (1991). Prog. Neuropathol.
Accordingly, respiratory chain and ATP synthase defects may result from genetic errors in either the nuclear or the mitochondrial genome. Thus, mitochondrial disorders may manifest as mendelian traits when caused by nDNA mutations, or as sporadic or maternally inherited traits when caused by mtDNA mutations. e. the coexistence of both normal and mutated mtDNA in the same tissue) is one factor responsible for the frequent observation of partial defects of respiratory chain enzymes. Traditional biochemistry has limitations in detecting such defects, as measurement of activity in homogenates gives an average value.
Ageing Dev. 43, 279-293. , Deadman, M. , Turnbull, D. , and Gardiner, R. M. (1991). Clin. Genet. 39, 33-38. Richter, C. (1988). FEBS Lett. 241, 1-5. , and Arnes, Β. Ν. (1988). Proc. Natl. Acad. Sei. A. 85, 64656467. , Saudubray, J. , and Munnich, A. (1988). Lancet 2, 567-568. , eds. (1991). "Mitochondrial Encephalomyopathies," Prog. , Vol. 7. Raven Press, New York. Satoh, M. , Rajewsky, M. , and Kuroki, T. (1988). /. Biol. Chem. 263, 68546856. Saul, R. , and Ames, Β. N. (1987). In "Modern Biological Theories of Aging" (H.
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